Foods oftentimes blamed for food allergies and sensitivity reactions are also believed to cause or lead to inflammatory and autoimmune conditions. These coarse foods are likely doing so straight through a process of gut inflammation resulting in leaky gut. This injury, especially occurring in genetically predisposed people, and in the setting of altered gut bacteria (dysbiosis), and immune stress likely predisposes to added inflammation and leaky gut. This vicious cycle is thought to allow toxic food protein-bacteria complexes to enter the body resulting in a variety of inflammatory and/or autoimmune conditions such as rheumatoid arthritis. A new study sheds some added light on link of food intolerance to rheumatoid arthritis is reviewed in this context.
Researchers from Norway in 2006 published in the British journal Gut added new evidence of the link in the middle of foods and rheumatoid arthritis. Professor Bradtzaeg and his colleagues at the construct of analysis in Oslo measured IgG, IgA, and IgM antibodies to foods. The measured these antibodies in blood and intestinal fluid in habitancy with rheumatoid arthritis compared with salutary people.
The researchers performed blood and intestinal fluid antibody tests to the following food antigens: gliadin, oats, cow's milk proteins (casein, lactalbumin, lactoglobulin), soy, pork, cod fish, and egg (ovalbumin). These foods are in the top 10 of coarse food allergens as well as food protein intolerances.
What they found was a "particularly astonishing (incidence) of cross reactive food antibodies in proximal gut secretions" as well as increased IgM antibodies to some of these foods in the blood. The findings in the blood were less astonishing than in the intestinal secretions. This is consistent with difficulties looking elevated blood antibodies to foods in habitancy with rheumatoid arthritis and other autoimmune/inflammatory conditions despite a lot of anecdotal and elimination diet perceive supporting the role of foods in these conditions. Interestingly, Dr. Ken Fine's stool antibodies tests may be on to something.
The results, in their opinion, indicate that measuring blood antibodies to foods in rheumatoid arthritis provides petite facts about the role of foods in rheumatoid arthritis. However, intestinal antibodies not only show a "striking" pattern of elevation consistent with adverse food immune reactions but also that there appears to be a inherent cumulative follow of manifold foods. That is, not only may some foods trigger an abnormal immune response resulting in joint inflammation but the aggregate of manifold question foods may be a key component to this link. Their results support the connection of mucosal (gut) immune activation from cross reaction of foods to rheumatoid arthritis in at least some people.
What might this mean? This data supports the thought and the perceive of many habitancy that elimination of distinct question food combinations may be useful in preventing or reducing joint inflammation. This is both enchanting and intriguing.
Multiple ordinarily eaten foods oftentimes linked to food allergies and sensitivities may be contributing to inflammatory and/or autoimmune conditions. These coarse question foods or their lectins are likely contributing to the process of gut inflammation. This is likely causing gut injury resulting in leaky gut. This injury and leaky gut, especially in genetically predisposed people, may, in the setting of altered gut bacteria (dysbiosis), predispose to added injury. This then allows the entry of toxic food protein (lectin)-bacteria complexes into the body, especially the blood stream. The follow is inflammatory and/or autoimmune conditions like rheumatoid arthritis.
This gut-joint axis is likely the same mechanism as the gut-brain axis and gut-skin axis that yield the myriad of symptoms and diseases we are now seeing. The linked food protein (lectin)-bacteria immune reactions in the gut are increasingly being blamed for the development of a myriad of diseases.
Much more needs to learned, but it is enchanting that distinct foods keep showing up as the usual suspects. These question foods or lectins include the grains (especially wheat, barley, rye, oats, corn), dairy (casein), nightshades (potato, tomato, peppers) and peanuts, soy and other legumes. Diets eliminating or restricting these foods have been reported as being useful for many symptoms and diseases. However, definitive links are difficult to construct because of limitations of scientific research.
The foods implicated are commonly petite in some manner in a variety of elimination diets such as the gluten-free/casein free diet, naked diet, paleolithic/hunter-gatherer or caveman diets, arthritis diet, low carbohydrate diet, anti-inflammatory diet, and six food elimination diet.
The Paleolithic or Hunter-Gatherer diet specifically recommends restricting grains, dairy and legumes. Assorted anti-inflammatory or arthritis diets commonly recommend eliminating whether wheat or gluten, dairy and the nightshades. The dietary approach to autism ordinarily advocated is a casein-free, gluten-free diet.
Despite lay collective reports of great successes with such elimination diets, mainstream treatment continues to be slow to study the dietary treatment of disease. However, especially in the past two to three years more studies are appearing showing links supporting a needful role of food and bacteria in the gut and Assorted autoimmune diseases.
